Ons. J Neurosci. 1994; fourteen:1123. [PubMed: 7509863] 61. Hajimohammadreza I, Raser KJ, Nath R, et al. Neuronal nitric oxide synthase and calmodulindependent protein kinase IIalpha go through neurotoxin-induced proteolysis. J Neurochem. 1997; sixty nine:10063. [PubMed: 9282922] sixty two. Hrabetova S, Sacktor TC. Bidirectional regulation of protein kinase M zeta in the servicing of long-term potentiation and long-term despair. J Neurosci. 1996; sixteen:53243. [PubMed: 8757245] 63. Wu HY, Tomizawa K, Oda Y, et al. Important function of calpain-mediated cleavage of calcineurin in excitotoxic neurodegeneration. J Biol Chem. 2004; 279:49290. [PubMed: 14627704] 64. Denny JB, Polan-Curtain J, Rodriguez S, et al. Proof that protein kinase M will not retain long-term potentiation. Brain Res. 1990; 534:201. [PubMed: 2073582] sixty five. Malinow R, Schulman H, Tsien RW. Inhibition of postsynaptic PKC or CaMKII blocks induction but not expression of LTP. Science. 1989; 245:862. [PubMed: 2549638] sixty six. Otmakhov N, Griffith LC, Lisman JE. Postsynaptic inhibitors of calciumcalmodulin-dependent protein kinase form II block induction although not maintenance of pairing-induced long-term potentiation. J Neurosci. 1997; seventeen:53575. [PubMed: 9204920] 67. Hu GY, Hvalby O, Walaas SI, et al. Protein kinase C injection into hippocampal pyramidal cells elicits capabilities of long-term potentiation. Nature. 1987; 328:426. [PubMed: 3614346] 68. Lledo PM, Hjelmstad GO, 135558-11-1 site Mukherji S, et al. Calciumcalmodulin-dependent kinase II and longterm potentiation improve synaptic transmission with the exact mechanism. Proc Natl Acad Sci Usa. 1995; 92:11175. [PubMed: 7479960] sixty nine. Xu L, Deng X. Suppression of most cancers cell migration and invasion by protein phosphatase 2A via dephosphorylation of mu-and m-calpains. J Biol Chem. 2006; 281:355675. [PubMed: 16982626] 70. 141430-65-1 medchemexpress Tallant EA, Brumley LM, Wallace RW. Activation of the calmodulin-dependent phosphatase by a Ca2-dependent protease. Biochemistry. 1988; 27:22051. [PubMed: 2837285] 71. Baumgartel K, Mansuy IM. Neural functions of calcineurin in synaptic plasticity and memory. Find out Mem. 2012; 19:3754. [PubMed: 22904368] 72. Evergren E, Marcucci M, Tomilin N, et al. Amphiphysin is usually a component of clathrin coats formed during synaptic vesicle recycling at the lamprey big synapse. Website traffic. 2004; five:5148. [PubMed: 15180828] 73. Wigge P, McMahon HT. The amphiphysin relatives of proteins as well as their job in endocytosis at the synapse. Trends Neurosci. 1998; 21:3394. [PubMed: 9720601] seventy four. Zhang B, Zelhof AC. Amphiphysins: elevating the BAR for synaptic vesicle recycling and membrane dynamics. Bin-Amphiphysin-Rvsp Website traffic. 2002; three:4520. 75. Wu Y, Liang S, Oda Y, et al. Truncations of amphiphysin I by calpain inhibit vesicle endocytosis all through neural hyperexcitation. Embo J. 2007; 26:29810. [PubMed: 17541403] seventy six . Di Rosa G, Odrijin T, Nixon RA, et al. Calpain inhibitors: a 1184136-10-4 Autophagy remedy for Alzheimer’s sickness. J Mol Neurosci. 2002; 19:1351. To start with post suggesting the use of calpain inhibitors with the treatment of Alzheimer’s ailment. [PubMed: 12212771] seventy seven. Ando K, Kudo Y, Takahashi M. Negative regulation of neurotransmitter release by calpain: a attainable involvement of specific SNAP-25 cleavage. J Neurochem. 2005; 94:651. [PubMed: 15992386] 78. Grumelli C, Berghuis P, Pozzi D, et al. Calpain action contributes into the handle of SNAP-25 concentrations in neurons. Mol Cell Neurosci. 2008; 39:3143. [PubMed: 18721885] seventy nine. Bi X, Chen J, Baudry M. Developmental modifications in calpain exercise, GluR1 recepto.
Heme Oxygenase heme-oxygenase.com
Just another WordPress site
